MOLECULAR GENETICS LABORATORY
Ph.D. Students
Gargi Gouranga Nanda
Email: gargi.nanda@niser.ac.in
My city of origin is Mumbai. I've done my MSc in Biotechnology from Sardar Patel University and joined NISER in 2011 to pursue my doctoral work with Dr. Debasmita. My thesis specialization is in the genetic and physiological analysis of Fuchs Dystrophy. There is an immense scope in the area of corneal dystrophies and it therefore has peaked my interest in it. Elaborate discussions in lab with my thesis advisor and peers have improved my professional and personal development.
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After graduating in Zoology from Patna University, I have been a member of this lab since 2014. I'm currently working on the role proteostasis components in the progression of Pseudoexfoliation (PEX). The plethora of unexplored niches in PEX has bestowed the liberty to test various ideas which are seeded during our weekly group meetings. Apart from these intense project discussions, we also have our candid moments.
I am from Behrampur, Odisha and have done my Masters in Biotechnology from Ravenshaw University. I've been working with Dr. Debasmita since 2012. My thesis work is focused on an age related neurodegenerative disorder called Pseudoexfoliation Syndrome, which is a leading cause of secondary glaucoma. I intend to find the role of chaperones in the development of this disease.
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I belong to West Bengal and after passing my M.Sc. from Calcutta University, I have joined the lab in 2015. My thesis work is focused on the role and regulation of endothelial to mesenchymal transition inducing protein ZEB1, responsible for the progression of Fuchs Endothelial Corneal Dystrophy. We try to understand the molecular mechanisms of ZEB1 induced EndoMT and want to validate new drugs, which might recover the pathogenesis of the disease.
I hail from Visakhapatnam, Andhra Pradesh. I completed my M.Sc. in Biotechnology from Madurai Kamaraj University in April 2016 and joined this lab in August 2016. Pseudoexfoliation Glaucoma is a multifactorial age-related eye disorder with both genetic as well as epigenetic factors implicated in its causation and progression. I work on understanding the epigenetic role of candidate genes associated with this disorder.